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Due to the similarities between myostatin and GDF11, the actions of GDF11 are likely regulated by WFIKKN2, a large extracellular multidomain protein consisting of follistatin, immunoglobulin, protease inhibitor, and NTR domains.

GDF11 has been identified as a blood circulating factor that has the ability to reverse age-related cardiac hypertrophy in mice.

The database is a copy of the public record as it existed on the date of retrieval.

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The Mexico-Finland forestry cooperation, international influence on the forestry policy, impacts/effects of international cooperation, the ‘context-analysis’ of international cooperation, community forest management policy.

GDF11 gene expression and protein abundance decreases with age, and it shows differential abundance between young and old mice in parabiosis procedures, causing youthful regeneration of cardiomyocytes, a reduction in the brain natriuretic peptide (BNP) and in the atrial natriuretic peptide (ANP).

GDF11 also causes an increase in expression of SERCA-2, an enzyme necessary for relaxation during diastolic functions.

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The methodology was carried out through a context-specific impact study; it consisted of describing changes in Mexican society and the history of the forest policy with the aim of understand the contribution of the Finnish-Mexican cooperation.

The information was collected following 3 steps: 1) collecting historical data in Mexico and from the archive of Ministry of Foreign Affairs of Finland; 2) interviews with Finnish and Mexican forestry experts involved in the cooperation interventions and 3) validating by contrasting sources.